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Trees were constructed from these sequences with 100 full maximum likelihood bootstrap replicates (implemented in PHYML [14]), following either complete removal of recombinant sequence fragments or the division of recombinant sequences into their constituent fragments by a blinded fully exploratory screen for recombination using RDP3 [15]. The recombination screen was fully exploratory in that eve
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L antibodies made also rapidly clear to the clinicians that a reliable predictive factor for outcome was, in fact, lacking [3-7]. The introduction of K-RAS mutational status analysis allowed a reliable selection of resistant patients (i.e. those with mutated K-RAS). However not all K-RAS wildtype cases were also responders to anti-EGFR monoclonal antibodies. This observation made the need for furt
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Itada M, Kitagawa H, Igarashi K, Hirose S, Kanakubo Y: Polyamine lowered the hepatic lipid peroxide level in rats. Res Commun Chem Pathol Pharmacol 1988, 62:235-249. Merentie M, Uimari A, Pietil?M, Sinervirta R, Kein en TA, Veps nen J, Khomutov A, Grigorenko N, Herzig KH, J ne J, Alhonen L: Oxidative stress and inflammation in the pathogenesis of activated polyamine catabolism-induced acute pancr
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Harp PM, Hahn BH: Origins of HIV and the AIDS Pandemic. Cold Spring Harb Perspect Med 2011, 1:a006841. 3. Brennan CA, Bodelle P, Coffey R, Devare SG, Golden A, Harris B, Holzmayer V, Luk KC, Schochetman G, Swanson P, Yamaguchi J, Vallari A, Ndembi N, Ngansop C, Makamche F, Mbanya D, Gurtler LG, Zekeng L, Kaptue L, Hackett J Jr: The prevalence of diverse HIV-1 strains was stable in Cameroonian bloo
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One of the newly sequenced gag genes (BS72) was apparently derived through recombination between F2 and CRF36_cpx parental viruses, one of the nef genes was apparently derived through recombination between F and CRF22_01A1 parental viruses. The phylogenetic analysis of gag sequences derived from the Cameroonian samples further revealed four sequences (BS09, BS25, BS16 and BS42) situated on diverge
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L antibodies made also rapidly clear to the clinicians that a reliable predictive factor for outcome was, in fact, lacking [3-7]. The introduction of K-RAS mutational status analysis allowed a reliable selection of resistant patients (i.e. those with mutated K-RAS). However not all K-RAS wildtype cases were also responders to anti-EGFR monoclonal antibodies. This observation made the need for furt
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NAlveolar macrophages play a central role in lung defense [3,36,37]. The class A scavenger receptors (SRA) MARCO and SR-AI/II are expressed on alveolar macrophages and function in innate defenses against inhaled pathogens and particles [7,8,10,11,17]. However, large number of murine alveolar macrophages with SRA deficient are rarely available for in vitro studies. To further investigate the role o
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Surface expression of the macrophage-associated differentiation Ags was assessed by direct immunofluorescence (thick lines). ZK1 cells were incubated with FITC-labeled anti-mouse F4/80 or FITC-labeled anti-mouse CD11b. Mouse IgG2a and IgG2b were used as isotype controls (light lines). Staining of cells with FITC-labeled anti-mouse Ig compared to unstained cells detects the of cells expressing su